The role of protein dynamics in the evolution of new enzyme function

The role of protein dynamics in the evolution of new enzyme function

Play all audios:

Loading...

Enzymes must be ordered to allow the stabilization of transition states by their active sites, yet dynamic enough to adopt alternative conformations suited to other steps in their catalytic


cycles. The biophysical principles that determine how specific protein dynamics evolve and how remote mutations affect catalytic activity are poorly understood. Here we examine a 'molecular


fossil record' that was recently obtained during the laboratory evolution of a phosphotriesterase from Pseudomonas diminuta to an arylesterase. Analysis of the structures and dynamics of


nine protein variants along this trajectory, and three rationally designed variants, reveals cycles of structural destabilization and repair, evolutionary pressure to 'freeze out'


unproductive motions and sampling of distinct conformations with specific catalytic properties in bi-functional intermediates. This work establishes that changes to the conformational


landscapes of proteins are an essential aspect of molecular evolution and that change in function can be achieved through enrichment of preexisting conformational sub-states.


We thank D.S. Tawfik for stimulating discussions. C.J.J. thanks the Australian Research Council for a Future Fellowship (FT140101059) and Discovery Project (DP130102144). This research was


undertaken on the MX1 and MX2 beamlines at the Australian Synchrotron, Victoria, Australia. F.H. thanks the Biotechnology and Biological Sciences Research Council and European Research


Council (starting investigator grants). M.K. thanks the EU Innovative Training Network (ProSA) for a studentship. N.T. is funded as a Canadian Institutes of Health Research new investigator


and a Michael Smith Foundation of Health Research (MSFHR) career investigator. N.T. thanks Natural Sciences and Engineering Research Council of Canada Discovery Grant RGPIN 418262-12. A.M.B.


is funded as a National Health and Medical Research Senior Research Fellow (1022688). This work was supported by the Victorian Life Sciences Computation Initiative, an initiative of the


Victorian Government, Australia.


Eleanor Campbell and Miriam Kaltenbach: These authors contributed equally to this work.


Research School of Chemistry, Australian National University, Canberra, Australia


Eleanor Campbell, Galen J Correy, Paul D Carr, Emma K Livingstone, Livnat Afriat-Jurnou & Colin J Jackson


Michael Smith Laboratories, University of British Columbia, Vancouver, British Columbia, Canada


Department of Biochemistry, University of Cambridge, Cambridge, UK


Biomedicine Discovery Institute, Monash University, Clayton, Victoria, Australia


Department of Biochemistry and Molecular Biology, Monash University, Clayton, Victoria, Australia


Institut de Biologie Structurale, University Grenoble Alpes, Commissariat à l'Energie Atomique and Centre National de la Recherche Scientifique, Grenoble, France


E.C., M.K., G.J.C., P.D.C., B.T.P., E.K.L. and L.A.-J. performed experiments and analyzed results; A.M.B. and M.W. analyzed results; F.H. conceived the project, designed experiments and


analyzed results; N.T. conceived the project, designed experiments, analyzed results and wrote the manuscript; and C.J.J. conceived the project, designed experiments, performed experiments,


analyzed results and wrote the manuscript.


Supplementary Results, Supplementary Figures 1–7 and Supplementary Tables 1–3. (PDF 1751 kb)


Anyone you share the following link with will be able to read this content: