Expression of calcium binding proteins in the auditory brainstem pathway of the jaundiced gunn rat † 829

Expression of calcium binding proteins in the auditory brainstem pathway of the jaundiced gunn rat † 829

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Sensorineural hearing loss is one of the major sequelae of neonatal hyperbilirubinemia. Retrocochlear sites of lesions due to kernicterus are related to significant abnormalities in the wave latencies, interwave intervals, and amplitudes of the auditory brainstem response. In the present study, changes in the expression of the calcium binding proteins (CBP) parvalbumin and calbindin-D28k have been examined in the homozygous recessive jaundiced (jj) Gunn rat by light microscopic immunohistochemistry at 15-18 days postnatally. The density of immunoreactive staining for both CBPs was reduced in the cochlear nuclei and the superior olivary complex, particularly the superior paraolivary and medial superior olivary nuclei, in comparison to age-matched heterozygous (Nj) control rats. At both sites, the extent of the reduction in immunoreactivity was related to the severity of the clinical symptoms. By contrast, immunoreactive staining in other brainstem auditory(e.g., dorsal and ventral nuclei of the lateral lemniscus, inferior colliculus, medial geniculate body) and neighboring non-auditory structures was similar in jaundiced and control rats. The developmental retardation of CBPs coincides with the period of synaptic refinement in the auditory brainstem, and the inability to buffer intracellular calcium effectively may lead to bilirubin-induced neuronal cell death, possibly by inhibition of Ca2+/calmodulin-dependent kinase II activity. Supported by NIH NIDCD R01 DC00369 (SMS) and MERIT Award EY02191 (RFS). AUTHOR INFORMATION AUTHORS AND AFFILIATIONS * Otolaryngology, Neurology, Pediatrics, Physiology and Anatomy, Medical College of Virginia, Virginia Commonwealth University, Richmond, VA, USA Justin J. G. Broughton, David L. Galiani, Steven M. Shapiro & Robert F. Spencer Authors * Justin J. G. Broughton View author publications You can also search for this author inPubMed Google Scholar * David L. Galiani View author publications You can also search for this author inPubMed Google Scholar * Steven M. Shapiro View author publications You can also search for this author inPubMed Google Scholar * Robert F. Spencer View author publications You can also search for this author inPubMed Google Scholar RIGHTS AND PERMISSIONS Reprints and permissions ABOUT THIS ARTICLE CITE THIS ARTICLE Broughton, J., Galiani, D., Shapiro, S. _et al._ Expression of Calcium Binding Proteins in the Auditory Brainstem Pathway of the Jaundiced Gunn Rat † 829. _Pediatr Res_ 41 (Suppl 4), 141 (1997). https://doi.org/10.1203/00006450-199704001-00849 Download citation * Issue Date: 01 April 1997 * DOI: https://doi.org/10.1203/00006450-199704001-00849 SHARE THIS ARTICLE Anyone you share the following link with will be able to read this content: Get shareable link Sorry, a shareable link is not currently available for this article. Copy to clipboard Provided by the Springer Nature SharedIt content-sharing initiative

Sensorineural hearing loss is one of the major sequelae of neonatal hyperbilirubinemia. Retrocochlear sites of lesions due to kernicterus are related to significant abnormalities in the wave


latencies, interwave intervals, and amplitudes of the auditory brainstem response. In the present study, changes in the expression of the calcium binding proteins (CBP) parvalbumin and


calbindin-D28k have been examined in the homozygous recessive jaundiced (jj) Gunn rat by light microscopic immunohistochemistry at 15-18 days postnatally. The density of immunoreactive


staining for both CBPs was reduced in the cochlear nuclei and the superior olivary complex, particularly the superior paraolivary and medial superior olivary nuclei, in comparison to


age-matched heterozygous (Nj) control rats. At both sites, the extent of the reduction in immunoreactivity was related to the severity of the clinical symptoms. By contrast, immunoreactive


staining in other brainstem auditory(e.g., dorsal and ventral nuclei of the lateral lemniscus, inferior colliculus, medial geniculate body) and neighboring non-auditory structures was


similar in jaundiced and control rats. The developmental retardation of CBPs coincides with the period of synaptic refinement in the auditory brainstem, and the inability to buffer


intracellular calcium effectively may lead to bilirubin-induced neuronal cell death, possibly by inhibition of Ca2+/calmodulin-dependent kinase II activity. Supported by NIH NIDCD R01


DC00369 (SMS) and MERIT Award EY02191 (RFS). AUTHOR INFORMATION AUTHORS AND AFFILIATIONS * Otolaryngology, Neurology, Pediatrics, Physiology and Anatomy, Medical College of Virginia,


Virginia Commonwealth University, Richmond, VA, USA Justin J. G. Broughton, David L. Galiani, Steven M. Shapiro & Robert F. Spencer Authors * Justin J. G. Broughton View author


publications You can also search for this author inPubMed Google Scholar * David L. Galiani View author publications You can also search for this author inPubMed Google Scholar * Steven M.


Shapiro View author publications You can also search for this author inPubMed Google Scholar * Robert F. Spencer View author publications You can also search for this author inPubMed Google


Scholar RIGHTS AND PERMISSIONS Reprints and permissions ABOUT THIS ARTICLE CITE THIS ARTICLE Broughton, J., Galiani, D., Shapiro, S. _et al._ Expression of Calcium Binding Proteins in the


Auditory Brainstem Pathway of the Jaundiced Gunn Rat † 829. _Pediatr Res_ 41 (Suppl 4), 141 (1997). https://doi.org/10.1203/00006450-199704001-00849 Download citation * Issue Date: 01 April


1997 * DOI: https://doi.org/10.1203/00006450-199704001-00849 SHARE THIS ARTICLE Anyone you share the following link with will be able to read this content: Get shareable link Sorry, a


shareable link is not currently available for this article. Copy to clipboard Provided by the Springer Nature SharedIt content-sharing initiative