Self-reactive mait cells are common in healthy individuals

Self-reactive mait cells are common in healthy individuals

Play all audios:

Loading...

Access through your institution Buy or subscribe Mucosal-associated invariant T (MAIT) cells have an almost invariant TCRα chain that is paired with a restricted repertoire of TCRβ chains and recognize specific microbial metabolites presented by the MHC class I-like molecule MR1. However, some MAIT cells also bind to non-microbial ligands, and alterations in MAIT cells have been observed in non-microbial inflammatory diseases such as diabetes, multiple sclerosis and obesity. Chancellor et al. show that a discrete population of circulating MAIT cells in healthy individuals has dual reactivity, binding both microbial antigens and self-antigens, and they identify a motif in the TCRβ chain that can facilitate such promiscuous MR1 binding. The authors propose that self-reactivity may depend on particular ligand-dependent conformations of MR1 rather than the direct interaction with MR1-bound ligand. Furthermore, they hypothesize that self-recognition by MAIT cells might have regulatory and/or homeostatic functions, and that self-reactive MAIT cells participate in inflammatory and autoimmune conditions with perturbed immune homeostasis, as well as in cancer surveillance and progression. This is a preview of subscription content, access via your institution ACCESS OPTIONS Access through your institution Access Nature and 54 other Nature Portfolio journals Get Nature+, our best-value online-access subscription $32.99 / 30 days cancel any time Learn more Subscribe to this journal Receive 12 print issues and online access $209.00 per year only $17.42 per issue Learn more Buy this article * Purchase on SpringerLink * Instant access to full article PDF Buy now Prices may be subject to local taxes which are calculated during checkout ADDITIONAL ACCESS OPTIONS: * Log in * Learn about institutional subscriptions * Read our FAQs * Contact customer support REFERENCES ORIGINAL ARTICLE * Chancellor, A. et al. Promiscuous recognition of MR1 drives self-reactive mucosal-associated invariant T cell responses. _J. Exp. Med._ 220, e20221939 (2023) Article  CAS  PubMed  Google Scholar  Download references AUTHOR INFORMATION AUTHORS AND AFFILIATIONS * Nature Reviews Immunology http://www.nature.com/nri/ Alexandra Flemming Authors * Alexandra Flemming View author publications You can also search for this author inPubMed Google Scholar CORRESPONDING AUTHOR Correspondence to Alexandra Flemming. RIGHTS AND PERMISSIONS Reprints and permissions ABOUT THIS ARTICLE CITE THIS ARTICLE Flemming, A. Self-reactive MAIT cells are common in healthy individuals. _Nat Rev Immunol_ 23, 543 (2023). https://doi.org/10.1038/s41577-023-00929-y Download citation * Published: 28 July 2023 * Issue Date: September 2023 * DOI: https://doi.org/10.1038/s41577-023-00929-y SHARE THIS ARTICLE Anyone you share the following link with will be able to read this content: Get shareable link Sorry, a shareable link is not currently available for this article. Copy to clipboard Provided by the Springer Nature SharedIt content-sharing initiative

Access through your institution Buy or subscribe Mucosal-associated invariant T (MAIT) cells have an almost invariant TCRα chain that is paired with a restricted repertoire of TCRβ chains


and recognize specific microbial metabolites presented by the MHC class I-like molecule MR1. However, some MAIT cells also bind to non-microbial ligands, and alterations in MAIT cells have


been observed in non-microbial inflammatory diseases such as diabetes, multiple sclerosis and obesity. Chancellor et al. show that a discrete population of circulating MAIT cells in healthy


individuals has dual reactivity, binding both microbial antigens and self-antigens, and they identify a motif in the TCRβ chain that can facilitate such promiscuous MR1 binding. The authors


propose that self-reactivity may depend on particular ligand-dependent conformations of MR1 rather than the direct interaction with MR1-bound ligand. Furthermore, they hypothesize that


self-recognition by MAIT cells might have regulatory and/or homeostatic functions, and that self-reactive MAIT cells participate in inflammatory and autoimmune conditions with perturbed


immune homeostasis, as well as in cancer surveillance and progression. This is a preview of subscription content, access via your institution ACCESS OPTIONS Access through your institution


Access Nature and 54 other Nature Portfolio journals Get Nature+, our best-value online-access subscription $32.99 / 30 days cancel any time Learn more Subscribe to this journal Receive 12


print issues and online access $209.00 per year only $17.42 per issue Learn more Buy this article * Purchase on SpringerLink * Instant access to full article PDF Buy now Prices may be


subject to local taxes which are calculated during checkout ADDITIONAL ACCESS OPTIONS: * Log in * Learn about institutional subscriptions * Read our FAQs * Contact customer support


REFERENCES ORIGINAL ARTICLE * Chancellor, A. et al. Promiscuous recognition of MR1 drives self-reactive mucosal-associated invariant T cell responses. _J. Exp. Med._ 220, e20221939 (2023)


Article  CAS  PubMed  Google Scholar  Download references AUTHOR INFORMATION AUTHORS AND AFFILIATIONS * Nature Reviews Immunology http://www.nature.com/nri/ Alexandra Flemming Authors *


Alexandra Flemming View author publications You can also search for this author inPubMed Google Scholar CORRESPONDING AUTHOR Correspondence to Alexandra Flemming. RIGHTS AND PERMISSIONS


Reprints and permissions ABOUT THIS ARTICLE CITE THIS ARTICLE Flemming, A. Self-reactive MAIT cells are common in healthy individuals. _Nat Rev Immunol_ 23, 543 (2023).


https://doi.org/10.1038/s41577-023-00929-y Download citation * Published: 28 July 2023 * Issue Date: September 2023 * DOI: https://doi.org/10.1038/s41577-023-00929-y SHARE THIS ARTICLE


Anyone you share the following link with will be able to read this content: Get shareable link Sorry, a shareable link is not currently available for this article. Copy to clipboard Provided


by the Springer Nature SharedIt content-sharing initiative