Every shared epitope allele for itself?

Every shared epitope allele for itself?

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What is the relationship between genetic and environmental risk factors in predisposing for rheumatoid arthritis (RA)? A new study provides more details on the interaction between the HLA


DRB1 shared epitope (SE) alleles and smoking in conferring the risk of autoantibody-positive RA. Access through your institution Buy or subscribe This is a preview of subscription content,


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OPTIONS: * Log in * Learn about institutional subscriptions * Read our FAQs * Contact customer support REFERENCES * Schellekens, G. A., de Jong, B. A., van den Hoogen, F. H., van de Putte,


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Rheum._ 60, 916–923 (2009). Article  PubMed  Google Scholar  Download references AUTHOR INFORMATION AUTHORS AND AFFILIATIONS * Leiden University Medical Center, Leiden, The Netherlands Diane


van der Woude & Tom W. J. Huizinga Authors * Diane van der Woude View author publications You can also search for this author inPubMed Google Scholar * Tom W. J. Huizinga View author


publications You can also search for this author inPubMed Google Scholar CORRESPONDING AUTHOR Correspondence to Tom W. J. Huizinga. ETHICS DECLARATIONS COMPETING INTERESTS The authors


declare no competing financial interests. RIGHTS AND PERMISSIONS Reprints and permissions ABOUT THIS ARTICLE CITE THIS ARTICLE van der Woude, D., Huizinga, T. Every shared epitope allele for


itself?. _Nat Rev Rheumatol_ 5, 477–478 (2009). https://doi.org/10.1038/nrrheum.2009.174 Download citation * Issue Date: September 2009 * DOI: https://doi.org/10.1038/nrrheum.2009.174 SHARE


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